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At the receptor level, luteolin’s differential binding to ER-α and ER-β allows it to function as a weak phytoestrogenic selective estrogen receptor modulator (SERM), displaying partial agonist activity in estrogen-deficient environments and antagonistic effects under estrogen-excess conditions (Maximov et al., 2013)
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Back Mojsov, S, Heinrich, G, Wilson, IB, et al, (5 Sept 1986) 'Preproglucagon gene expression in pancreas and intestine diversifies at the level of post-translational processing', The Journal of Biological Chemistry , 261/25, 11880-89
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