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While induced metabolic alkalosis would, in theory, be very effective with respect to performance during activities causing extensive perturbations in acid–base balance by reducing the rate of developing muscular fatigue by delaying the decrease in intracellular pH and enhancing muscle energy supply through glycolysis [93], the metabolic alkalosis induced through sodium citrate ingestion did not improve UBISP in the experimental group, which may suggest that the degree of alkalosis was insufficient for enhancing glycolytic ATP production
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