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It was shown in macrophages that IFN- γ signaling can induce PGC1 β -dependent upregulation of ERR α to promote the generation of mitochondrial-derived reactive oxygen species
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Sodium dichloroacetate (DCA) An analysis of 200 ME/CFS patients and 102 healthy individuals revealed reduced amino acids involved in oxidative metabolism, indicative of PDH dysfunction [76]
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