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Mitophagy is critical for cellular homeostasis, as insufficient mitophagy can lead to the release of mitochondrial components, such as mtDNA, into the cytoplasm, triggering inflammation [29]
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Following brain injury, CD4⁺ T cells differentiate into various effector subsets, each of which secretes distinct cytokines that exert either proinflammatory or anti-inflammatory effects [157, 158]
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