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KPV exerts its primary anti-inflammatory activity through direct intracellular penetration of immune cells and intestinal epithelial cells, where it suppresses NF-κB transcription factor activity — the master regulator of pro-inflammatory cytokine gene expression — through a receptor-independent intracellular mechanism, resulting in downstream reduction of IL-6, IL-8, TNF-α, and related inflammatory mediators

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Interest in tissue-repair mechanisms has contributed to the growing popularity of BPC-157 Peptide within recovery and regenerative medicine discussions